MS-theory
Theory
Our theory is founded on the conviction that MS is the result of a lack of neurotransmitters and caused by several factors.
This shortage can be accentuated by:
- Metal toxaemia (e.g. amalgam, welding, etc.)
- Genetic factors
- Psychological factors (often trigger factors) stress, demands and “musts”.
Background
Nerve signals are in part transmitted electrically via nerve fibre and in part bio-chemically via nerve junctions (synapses). The bio-chemical changeover is handled via neurotransmitters. With MS, the casing (myelin) around the nerve fibre is damaged in some places, although the nerve itself is generally undamaged. Scar tissue in the myelin allows the nerve signal to “leak out” and results in too little of the original signal being transmitted. My theory is that if it is possible to amplify the signal then a stronger signal will reach its destination despite the leakage from the myelin sheath. The signal can be amplified using medicines with an effect on neurotransmitters.
It is clear that the neurotransmitters must exist in a given relationship to each other for the nerve signals to travel correctly. When this relationship is altered it results in neurological symptoms. These frequently manifest themselves in numbness or other sensory disturbances, vision problems, fatigue, lack of strength, constipation/diarrhoea, a frequent need to urinate or difficulty in evacuating the bladder which is often accompanied by residual urine.
Given the above it is understandable that treatment must be related to variations in the levels of the different neurotransmitters.
Initially, different patients have shortages of different neurotransmitters and consequently must be treated according to an individually tailored program. No side affects occur when the medications are taken as prescribed. The patient ingests substances that are in short supply. This is comparable to a diabetic who takes the correct amount of insulin and does not suffer any side effects. My experience suggests that after a period of medication, the body can provide the needed increase in neurotransmitter production, and consequently the use of medicines can be gradually eliminated. At this stage I generally recommend an increase in the consumption of a variety of amino acids, which are the building blocks of neurotransmitters. Normal neurotransmitter production is not sufficient. A higher level of neurotransmitters is essential to amplify the nerve impulses so they can pass through the nerve fibre despite the damaged myelin sheath. The patient also needs to conserve the neurotransmitters that exist since they are required for all bodily activities including thinking, movement, anxiety and stress. It is possible to preserve neurotransmitters by resting a great deal and by engaging in activities that heighten well-being. I believe, for example, that it is a shame and unnecessary when patients deplete their small stock of neurotransmitters by climbing stairs, engaging in physical training and even making beds for their family members.
When one has reached a reasonable level of well-being via medicines and rest there is a temptation to start living a “normal” life. You wake up one morning and feel more energetic than you have for a long time and the ambitious ego starts doing all of the things that previously had to wait. The backlash usually comes quickly and relentlessly. I generally warn of this but it appears that everyone has to learn from personal experience. Lifestyle changes take a long time, and new boundaries must be tested with extreme caution. Preferably, one should avoid even approaching the limits. Everything is allowed providing it does not increase symptoms, neither in the short run, nor in the long.
There is a strong link between depression and MS. Depression does not appear after being diagnosed with MS; it usually comes first. My clinical experience shows that many MS sufferers have had an unusually troublesome or unpleasant period before the debut of the disease.
We also know that in depressive states the levels of neurotransmitters (primarily noradrenaline and serotonin) decline. It is also known that immune reactions appear during depression. It has recently been established that a shortage of e.g. noradrenaline can also lead to the occurrence of inflammatory reactions in the brain.
Earlier research also indicates that a reduction has taken place in several of the nervous system's neurotransmitters, particularly noradrenaline and serotonin, in more severe MS cases. Recent research has also indicated a dysfunction in the neurotransmitter acetylcholine. This can result in deficits in the cognitive domains of memory, learning, attention and information processing In other words, the medical aspect of this treatment aims to amplify the nerve impulses by compensating for low neurotransmitter levels or neurotransmitters out of balance.
Mercury (Hg80)
According to WHO, amalgam fillings are the greatest source of mercury in the body. Amalgam contains about 50% mercury (Hg). Toxic mercury vapours are continually released from amalgam fillings. Mercury vapours are one of the most powerful known neurotoxins.
- Mercury hinders the production of neurotransmitters. As a result, the transmission of nerve impulses may be hampered, made impossible or “go wrong”. Symptoms such as the loss of sensitivity, numbness, prickling, tingling etc can arise.
- Mercury increases the quantity of free radicals, which in turn lead to oxidation in the body, particularly if there is a shortage of antioxidants.
- Mercury also settles on sulphur and hydrogen groups, thus disturbing the function of enzymes and membranes.
- Mercury induces autoimmun responses.
NOTE!
If you are considering replacing your amalgam fillings with plastic (composite) or ceramic fillings, you must obtain KNOWLEDGE before starting the procedure. During replacement, the released quantity of Hg is unavoidably higher and you risk more serious symptoms. Contact a dentist with lengthy experience in the procedure (particularly with MS patients) and, above all, do the procedure in collaboration with a PHYSICIAN who is knowledgeable and understands the problems that can arise.
Contact your local dental association for more information on the replacement of amalgam fillings.